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Mazor, R., et al. “Mesenteric Fat Cryolipolysis Attenuates Insulin Resistance In The Ossabaw Swine Model Of The Metabolic Syndrome.”. Surgery For Obesity And Related Diseases : Official Journal Of The American Society For Bariatric Surgery.
Balikcioglu, P. G., et al. “Branched-Chain Α-Keto Acids And Glutamate/Glutamine: Biomarkers Of Insulin Resistance In Childhood Obesity.”. Endocrinology, Diabetes & Metabolism.
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Hart, P. A., et al. “A Reduced Pancreatic Polypeptide Response Is Associated With New Onset Pancreatogenic Diabetes Versus Type 2 Diabetes.”. The Journal Of Clinical Endocrinology And Metabolism.
Chang, C. A., et al. “Curative Islet And Hematopoietic Cell Transplantation In Diabetic Mice Without Toxic Bone Marrow Conditioning.”. Cell Reports, p. 111615.
Shrestha, N., et al. “Integration Of Er Protein Quality Control Mechanisms Defines Β-Cell Function And Er Architecture.”. The Journal Of Clinical Investigation.
Moore, T. M., et al. “Parkin Regulates Adiposity By Coordinating Mitophagy With Mitochondrial Biogenesis In White Adipocytes.”. Nature Communications, p. 6661.
Dickerson, M. T., et al. “G Protein-Coupled Receptor Inhibition Of Beta-Cell Electrical Excitability And Insulin Secretion Depends On Na/K Atpase Activation.”. Nature Communications, p. 6461.
Du, W., et al. “Pharmacological Conversion Of Gut Epithelial Cells Into Insulin-Producing Cells Lowers Glycemia In Diabetic Animals.”. The Journal Of Clinical Investigation.
Hepler, C., et al. “Time-Restricted Feeding Mitigates Obesity Through Adipocyte Thermogenesis.”. Science (New York, N.y.), pp. 276-284.
Park, J., et al. “Activation Of The Insulin Receptor By An Insulin Mimetic Peptide.”. Nature Communications, p. 5594.